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- Impaired LEF1 Activation Accelerates iPSC-Derived Keratinocytes Differentiation in Hutchinson-Gilford Progeria Syndrome
- The Potentials of Methylene Blue as an Anti-Aging Drug
- Gene targeting techniques for Huntington’s disease
- Mechanisms of angiogenic incompetence in Hutchinson-Gilford progeria via downregulation of endothelial NOS
- Ultraviolet radiation protection potentials of Methylene Blue for human skin and coral reef health
- A targeted antisense therapeutic approach for Hutchinson-Gilford progeria syndrome
- In vivo base editing rescues Hutchinson–Gilford progeria syndrome in mice
- Peroxisomal abnormalities and catalase deficiency in Hutchinson-Gilford Progeria Syndrome.
- iPSC-derived endothelial Cells Affect Vascular Function in a Tissue Engineered Blood Vessel Model of Hutchinson-Gilford Progeria Syndrome.
- Transient induction of telomerase expression mediates senescence and reduces tumorigenesis in primary fibroblasts.
- Diminished Canonical β‐Catenin Signaling During Osteoblast Differentiation Contributes to Osteopenia in Progeria
- Comprehensive map of age-associated splicing changes across human tissues and their contributions to age-associated disease.
- Chromatin and Genomic determinants of alternative splicing
- A tissue engineered blood vessel model of Hutchinson-Gilford Progeria syndrome using Human iPSC-derived smooth muscle cells
- Lamin A and microtubules collaborate to maintain nuclear morphology
- Anti-Aging Potentials of Methylene Blue for Human Skin Longevity
- Lamin A and microtubules collaborate to maintain nuclear morphology
- Comparing lamin proteins post-translational relative stability using a 2A peptide-based system reveals elevated resistance of progerin to cellular degradation
- Loss of H3K9me3 Correlates with ATM Activation and Histone H2AX Phosphorylation Deficiencies in Hutchinson-Gilford Progeria Syndrome
- Mechanisms of genome instability in Hutchinson-Gilford progeria
- Methylene blue alleviates nuclear and mitochondrial abnormalities in progeria
- Phenotype-Dependent Coexpression Gene Clusters: Application to Normal and Premature Ageing
- Mechanisms controlling the smooth muscle cell death in progeria via down-regulation of poly(ADP-ribose) polymerase 1
- Nuclear localization signal deletion mutants of lamin A and progerin reveal insights into lamin A processing and emerin targeting
- From Cellular Characteristics to Disease Diagnosis: Uncovering Phenotyps with Supercells.
- An inhibitory role of progerin in the gene induction network of adipocyte differentiation from iPS cells
- An inhibitory role of progerin in the gene induction network of adipocyte differentiation from iPS cells
- Mouse Models of Laminopathies.
- Progeria: Translational Insights from Cell Biology.
- Cellular Basis of Laminopathies
- Correlated Alterations in Genome Organization, Histone Methylation, and DNA-Lamin A/C Interactions in Hutchinson-Gilford Progeria Syndrome.
- Automated image analysis of nuclear shape: what can we learn from a prematurely aged cell?
- Rapamycin activates autophagy in Hutchinson-Gilford Progeria Syndrome: Implications for normal aging and age-dependent neurodegenerative disorders.
- Rapamycin reverses cellular phenotype and enhances mutant protein clearance in Hutchinson Gilford progeria syndrome.
- Progerin and telomere dysfunction collaborate to trigger cellular senescence in normal human fibroblasts
- Cardiovascular Pathology in Hutchinson-Gilford Progeria: Correlation With the Vascular Pathology of Aging.
- A farnesyltransferase inhibitor prevents both the onset and late progression of cardiovascular disease in a progeria mouse model.
- A lamin A protein isoform overexpressed in Hutchinson-Gilford progeria syndrome interferes with mitosis in progeria and normal cells.
- Chromosome alignment and segregation regulated by ubiquitination of survivin
- The Cdc48/p97-Ufd1-Npl4 Complex: Its Potential Role in Coordinating Cellular Morphogenesis During the M-G (1) Transition
- Ran in the Spindle Checkpoint: A New Function for A Versatile GTPase
- The AAA-ATPase Cdc48/p97 Regulates Spindle Disassembly at the End of Mitosis
- A Ran Signaling Pathway Mediated by the Mitotic Kinase Aurora A in Spindle Assembly
- Characterization and Reconstitution of Drosophila gamma-Tubulin Ring Complex Subunits
