Progerin and telomere dysfunction collaborate to trigger cellular senescence in normal human fibroblastsPost author:caolab75@gmail.comPost published:June 1, 2011Post category:ArticlesJCI43578 You Might Also Like In vivo base editing rescues Hutchinson–Gilford progeria syndrome in mice January 6, 2021 Mechanisms of genome instability in Hutchinson-Gilford progeria November 10, 2016 Rapamycin reverses cellular phenotype and enhances mutant protein clearance in Hutchinson Gilford progeria syndrome. June 29, 2011
Rapamycin reverses cellular phenotype and enhances mutant protein clearance in Hutchinson Gilford progeria syndrome. June 29, 2011
