Skip to content
Cao Lab
  • Research
    • Current Research
    • What is Progeria?
    • Publications
  • Contact
  • Meet The Team
Menu Close
  • Research
    • Current Research
    • What is Progeria?
    • Publications
  • Contact
  • Meet The Team

Loss of H3K9me3 Correlates with ATM Activation and Histone H2AX Phosphorylation Deficiencies in Hutchinson-Gilford Progeria Syndrome

  • Post author:caolab75@gmail.com
  • Post published:December 1, 2016
  • Post category:Articles
journal.pone_.0167454

You Might Also Like

Mechanisms controlling the smooth muscle cell death in progeria via down-regulation of poly(ADP-ribose) polymerase 1

April 10, 2014

Nuclear localization signal deletion mutants of lamin A and progerin reveal insights into lamin A processing and emerin targeting

February 2, 2014

Ultraviolet radiation protection potentials of Methylene Blue for human skin and coral reef health

May 28, 2021
Cao Lab

My laboratory would like to further examine the causative relationship between progerin production and the aging process, to study the regulation of progerin production in normal cells, and to determine the contribution of progerin in normal human aging.

  • What is Progeria?Opens in a new tab
  • Current ResearchOpens in a new tab
  • PublicationsOpens in a new tab
  • Meet The TeamOpens in a new tab
  • MBlue LabsOpens in a new tab
  • 2219 Bioscience Research Building
  • 301-405-3408
  • Email UsOpens in your application